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rcc cell lines 786o  (ATCC)


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    ATCC rcc cell lines 786o
    Rcc Cell Lines 786o, supplied by ATCC, used in various techniques. Bioz Stars score: 99/100, based on 2275 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
    https://www.bioz.com/product/rcc+cell+lines+786o/786-O/pm40897466-41-7-16
    Average 99 stars, based on 2275 article reviews
    rcc cell lines 786o - by Bioz Stars, 2026-10
    99/100 stars

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    Article Title: Cuprous oxide nanoparticles trigger ER stress-induced apoptosis by regulating copper trafficking and overcoming resistance to sunitinib therapy in renal cancer.
    Article Snippet: While the current standard first-line treatment for advanced renal cell carcinoma (RCC) is sunitinib, patients inevitably develop resistance to this drug.. However, the rapid development of nanotechnology has provided emerging techniques for the treatment of advanced tumours, including RCC.. In our previous research, cuprous oxide nanoparticles (CONPs) showed ideal anti-tumour effects and low systemic toxicity.



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    rcc cell lines 786o  (ATCC)
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    The impact of PIK3AP1 knockdown on renal clear cell carcinoma cells. A PIK3AP1 expression in kidney renal clear cell carcinoma (KIRC) tissues compared to normal tissues. B The results of CCK-8 assays. C Colony formation assays, where PIK3AP1 knockdown markedly decreased the number of colonies formed by <t>786O</t> and OS-RC-2 cells ( p < 0.01). D EdU incorporation assays, indicating a substantial reduction in DNA synthesis in PIK3AP1 knockdown cells. E migration assays, demonstrating that PIK3AP1 knockdown significantly impairs the migratory ability of both cell lines ( p < 0.01). F invasion assays, highlighting a significant reduction in the invasive capacity of PIK3AP1 knockdown cells ( p < 0.001)
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    cell culture human rcc cell lines 786o  (ATCC)
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    The impact of PIK3AP1 knockdown on renal clear cell carcinoma cells. A PIK3AP1 expression in kidney renal clear cell carcinoma (KIRC) tissues compared to normal tissues. B The results of CCK-8 assays. C Colony formation assays, where PIK3AP1 knockdown markedly decreased the number of colonies formed by <t>786O</t> and OS-RC-2 cells ( p < 0.01). D EdU incorporation assays, indicating a substantial reduction in DNA synthesis in PIK3AP1 knockdown cells. E migration assays, demonstrating that PIK3AP1 knockdown significantly impairs the migratory ability of both cell lines ( p < 0.01). F invasion assays, highlighting a significant reduction in the invasive capacity of PIK3AP1 knockdown cells ( p < 0.001)
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    The impact of PIK3AP1 knockdown on renal clear cell carcinoma cells. A PIK3AP1 expression in kidney renal clear cell carcinoma (KIRC) tissues compared to normal tissues. B The results of CCK-8 assays. C Colony formation assays, where PIK3AP1 knockdown markedly decreased the number of colonies formed by <t>786O</t> and OS-RC-2 cells ( p < 0.01). D EdU incorporation assays, indicating a substantial reduction in DNA synthesis in PIK3AP1 knockdown cells. E migration assays, demonstrating that PIK3AP1 knockdown significantly impairs the migratory ability of both cell lines ( p < 0.01). F invasion assays, highlighting a significant reduction in the invasive capacity of PIK3AP1 knockdown cells ( p < 0.001)
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    The impact of PIK3AP1 knockdown on renal clear cell carcinoma cells. A PIK3AP1 expression in kidney renal clear cell carcinoma (KIRC) tissues compared to normal tissues. B The results of CCK-8 assays. C Colony formation assays, where PIK3AP1 knockdown markedly decreased the number of colonies formed by 786O and OS-RC-2 cells ( p < 0.01). D EdU incorporation assays, indicating a substantial reduction in DNA synthesis in PIK3AP1 knockdown cells. E migration assays, demonstrating that PIK3AP1 knockdown significantly impairs the migratory ability of both cell lines ( p < 0.01). F invasion assays, highlighting a significant reduction in the invasive capacity of PIK3AP1 knockdown cells ( p < 0.001)

    Journal: Clinical and Experimental Medicine

    Article Title: The role of BUD31 in clear cell renal cell carcinoma: prognostic significance, alternative splicing, and tumor immune environment

    doi: 10.1007/s10238-024-01451-8

    Figure Lengend Snippet: The impact of PIK3AP1 knockdown on renal clear cell carcinoma cells. A PIK3AP1 expression in kidney renal clear cell carcinoma (KIRC) tissues compared to normal tissues. B The results of CCK-8 assays. C Colony formation assays, where PIK3AP1 knockdown markedly decreased the number of colonies formed by 786O and OS-RC-2 cells ( p < 0.01). D EdU incorporation assays, indicating a substantial reduction in DNA synthesis in PIK3AP1 knockdown cells. E migration assays, demonstrating that PIK3AP1 knockdown significantly impairs the migratory ability of both cell lines ( p < 0.01). F invasion assays, highlighting a significant reduction in the invasive capacity of PIK3AP1 knockdown cells ( p < 0.001)

    Article Snippet: The human renal cell carcinoma (RCC) cell lines 786O, OS-RC-2, ACHN, CAKI-1, A498, and 769P, as well as the human renal proximal tubular epithelial cell line HK2, were purchased from the American Type Culture Collection (ATCC).

    Techniques: Knockdown, Expressing, CCK-8 Assay, DNA Synthesis, Migration